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Next topic for discussion: Acute intermittent porphyria (AIP) #Ayurveda
Gary Yuen: I do not know
much about the condition. If it is a genetic factor that prevents haem
synthesis, I think it is worth looking at iron consumption. There is a study
associating cigarette smoking with attacks. Research suggests examining diet
although I do not know what parts of it are typically considered. There is one
study among Indians that shows X porpyrin excretion is significantly higher
among meat eaters. I can't find the DOI so I'm unable to find the article. https://www.ncbi.nlm.nih.gov/pubmed/28134151
There seem to be
several studies showing the effect of lowering protein intake, though their
results are varied.
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Dr. Vikas Sharma:
In ayurveda you can corelate this with advancement of grehni vyadhi.vataj
grehni spacialy
Italy Amadeo bian: I am very
interested in a possible ayurvedic interpretation and a possible therapy
Gary Yuen: It would be
some blood disorder, possibly related to excess?
Ayulink: Master Amadio,
can u give more details of this disease, patient details, symptoms etc? Please
share more details. I think this disease is not known in India.
Gary Yuen: Consensus seems
to be that it is a hereditary disorder though I don't know if some think it is
not and may only be a change in gene expression. Symptoms seem to occur only in
a small number of people so there are likely exogenous factors. Some mentioned
include toxins, drugs, alcohol, smoking, and diet. Some treatments mentioned
increasing carbohydrate intake, though I do not know if that means patients
tended to have higher meat consumption.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4562648/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4541194/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4563803/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3583083/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5473191/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4717930/
Ayulink: Thanks Gary
Yuen. This is great help. I wonder how can we correlate this condition with
Ayurveda
Gary Yuen: If I understand
what I've read so far correctly, it seems to be some iron metabolism disorder,
perhaps of blood creation, perhaps if excess iron, or something else in its
metabolism or elimination.
Vd. Dhruti
Kagrana: Acute intermittent porphyria (AIP) is a genetic metabolic disorder
affecting the production of heme, the oxygen-binding prosthetic group of
hemoglobin. It is characterized by a deficiency of the enzyme porphobilinogen
deaminase.
Acute
Intermittent Porphyria (AIP)
This is one of
the hereditary hepatic porphyrias. Its inheritance is autosomal dominant. The
deficient enzyme is porphobilinogen deaminase (PBGD), also known as
hydroxymethylbilane synthase (HMB synthase). This enzyme was formerly known as
uroporphyrinogen I-synthase, and this term is still used by some clinical
laboratories. A deficiency of PBGD is not sufficient by itself to produce AIP,
and other activating factors must also be present. These include hormones,
drugs and dietary changes. Sometimes, activating factors cannot be identified.
Symptoms
Most people who
inherit the gene for AIP never develop symptoms. However, experts recommend
that all relatives of someone with AIP obtain testing, to determine who has the
genetic trait and who does not. Those who test positive for the trait should be
educated as to measures that will help avoid attacks. Prevention is essential
to good management.
AIP manifests
after puberty, especially in women (due to hormonal influences). Symptoms
usually come as discrete attacks that develop over two or more days. Abdominal
pain, which is associated with nausea, can be severe and occurs in most cases.
Other symptoms
may include:
nausea
vomiting
constipation
pain in the back,
arms and legs
muscle weakness
(due to effects on nerves supplying the muscles)
urinary retention
palpitation (due
to a rapid heart rate and often accompanied by increased blood pressure)
confusion,
hallucinations and seizures
Sometimes the
level of salt (sodium and chloride) in the blood decreases markedly and
contributes to some of these symptoms. The skin is not affected.
http://www.porphyriafoundation.com/about-porphyria/types-of-porphyria/AIP
Shri Minoo
parabia: I would suggest the fresh decoction of mixture of Tribulus terrestris
fruits and Boerhavia diffusa roots, taken in equal proportion , taken orally.
Coarse ground
above mixture 10g is to be soaked in 200ml of water at night. Next morning boil
till 50ml remains. Strain and give the filtrate to drink.
Repeat daily.
Ayulink: If it is
dhatvagni dushti, pippali must work positively. Even if it is related with heme
or blood pathogens, pippali is good at penetration in micro level conditions.
Am I correct?
Vd. Jignesh
Thakkar: Vataj grahani symptoms. Use chitrak takra prayog mentioned in charak
samhita
Vd. Hardik bhatt: AIP .. can we
correlate with rakt- pitaa???
Dr. Vikas Sharma: In ayurveda you can corelate this with
advancement of grahni vyadhi.vataj grahni spacially.
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Compiled by - Dr. Dhruti
Kagrana
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